Published: 18 May 2021
Author(s): Michele Barone, Patrizia Suppressa, Maria Teresa Viggiani, Gennaro Mariano Lenato, Arnaldo Scardapane, Luigi Chiumarulo, Nicola Ambrosini, Alfredo Di Leo, Carlo Sabbà
Issue: August 2021
Section: Original article

Hepatic encephalopathy (HE) is a potentially reversible disorder characterized by neuropsychiatric abnormalities and motor disturbances ranging from mild alterations of cognitive and motor functions to coma and death [1,2]. This condition has been attributed to the combination of gut-derived toxins production, portosystemic shunts and liver dysfunction, leading to endotoxemia associated with systemic and cerebral inflammation [3]. The subclinical expression of HE is defined as minimal hepatic encephalopathy (mHE) [4].


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