Published: 23 November 2023
Author(s): Antonio De Vita, Isabella Bruno, Silvia Baroni, Giacomo Moretti, Federica Tempestini, Alessandro Telesca, Saverio Tremamunno, Tamara Felici, Alfonso Verrillo, Valeria Tempesta, Vanessa Feudo, Priscilla Lamendola, Giovanna Liuzzo, Filippo Crea, Alessandro Giordano, Gaetano Antonio Lanza
Issue: May 2024
Section: Original Article

The detection of increased serum levels of cardiac troponins in a clinical context of acute myocardial ischemia is a crucial finding for the diagnosis of acute myocardial infarction (AMI) [1,2]. However, troponins may increase in various conditions of myocardial injury different from ischemic myocardial necrosis (e.g. anemia, hypoxemia, renal failure, heart failure, sepsis, tachyarrhythmias, etc.) [3,4]. Furthermore, a few studies suggested that cardiac troponins might also increase simply following myocardial ischemia, in the absence of cell necrosis [5–11], also suggesting the possibility that a significant increase of troponins after stress testing might increase the diagnostic yield for obstructive coronary artery disease (CAD).


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