Published: 28 November 2019
Author(s): Massimo Francesco Piepoli, Elisabetta Salvioni, Ugo Corrà, Francesco Doni, Alice Bonomi, Rocco La Gioia, Giuseppe Limongelli, Stefania Paolillo, Gianfranco Sinagra, Angela B. Scardovi, Rosa Raimondo, Michele Emdin, Federica Re, Mariantonietta Cicoira, Michele Correale, Roberto Badagliacca, Francesco Clemenza, Carlo Lombardi, Piergiuseppe Agostoni
Section: Original article

The association between elevated serum uric acid (SUA) levels and risk of cardiovascular (CV) disease has gained a lot of attention over the last years, considering that using the cut-off levels derived from gout data (6 mg/dl in women and 7 mg/dl in men), the prevalence of hyperuricemia in general population ranges from 20 to 25% [1–3]. Many epidemiological studies reported that elevated SUA levels are a powerful predictor of adverse clinical outcomes in patients with hypertension, diabetes, ischemic heart disease, stroke, chronic kidney disease or heart failure (HF) [4–7].

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