Published: 17 August 2022
Author(s): Giuseppe De Luca, Angelo Silverio, Monica Verdoia, Zbigniew Siudak, Tomasz Tokarek, Thomas A. Kite, Anthony H. Gershlick, Oriol Rodriguez-Leor, Belen Cid-Alvarez, Daniel A. Jones, Krishnaraj S. Rathod, José M. Montero-Cabezas, Alfonso Jurado-Roman, Matteo Nardin, Gennaro Galasso
Section: Original article

Coronavirus disease 2019 (COVID-19) has strongly impacted on worldwide healthcare systems with significant resources diverted to deal with this unforeseeable modern pandemic. Several reports have described an increased cardiovascular mortality during the COVID-19 pandemic due to direct [1,2] and indirect effects of severe acute respiratory syndrome coronavirus 2 (SARS-CoV-2) infection [3–8]. COVID-19 may induce oxygen supply/demand imbalance, abnormal systemic inflammatory response, atherosclerotic plaque rupture, overactivation of the coagulation system, and platelet hyperreactivity [1,2].


Stay informed on our latest news!


This question is for testing whether or not you are a human visitor and to prevent automated spam submissions.