Published: 4 February 2021
Author(s): Laura Pasin, Giulio Cavalli, Paolo Navalesi, Nicolò Sella, Giovanni Landoni, Andrey G. Yavorovskiy, Valery V. Likhvantsev, Alberto Zangrillo, Lorenzo Dagna, Giacomo Monti
Issue: May 2021
Section: Original article

Severe acute respiratory syndrome coronavirus 2 (SARS-CoV-2) affected 104.956.439 people worldwide and caused the death of 2.290.488 as of February 7, 2121[1]. Life-threatening cases of the coronavirus disease 2019 (COVID-19) are typically characterized by a detrimental hyper-inflammatory host response to the virus, which is reminiscent of the cytokine storm which develops in macrophage activation syndrome or after chimeric antigen receptor T-cell treatment, with massive release of pro-inflammatory cytokines [2,3].

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