Published: 7 May 2024
Author(s): Riccardo M. Inciardi, Marco Merlo, Mariagiulia Bellicini, Martina Setti, Antonio De Luca, Nunzia Di Meo, Paolo Rondi, Matteo Pagnesi, Marianna Adamo, Carlo M. Lombardi, Jacopo Giulio Rizzi, Davide Farina, Alessandro Mantovani, Giovanni Targher, Gianfranco Sinagra, Marco Metra, NAFLD-DCM group
Section: Original Article

Liver damage frequently occurs in patients with cardiovascular (CV) disease and predicts worse clinical outcomes [1,2]. In this context, liver damage may be mainly caused by elevated hepatic venous pressures due to increased cardiac filling pressures or by non-alcoholic fatty liver disease (NAFLD) [1]. NAFLD (recently renamed as metabolic dysfunction-associated fatty liver disease [MAFLD]) has become the most common chronic liver disease worldwide, affecting almost a third of the general adult population and up to ∼70 %−90 % of patients with type 2 diabetes mellitus or severe obesity [3,4].

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