Etiopathogenesis of cardiac dysfunction in septic shock is multifactorial, arising from autonomic dysregulation, increased capillary permeability, metabolic and mitochondrial dysfunction, oxidative stress, and activation of cytokines and inflammatory factors [1,2]. Transthoracic echocardiography is the main tool to early diagnose cardiac dysfunction in septic shock patients [3], but the use of complementary tools to improve the diagnose and treatment of these patients is much warranted.