Published: 4 October 2021
Author(s): Cristina Murcia-Gubianas, Juan Carlos Yébenes, Maria Buxó, Sara Foradada, Elisabeth Pinart
Issue: January 2022
Section: Letter to the Editor

Etiopathogenesis of cardiac dysfunction in septic shock is multifactorial, arising from autonomic dysregulation, increased capillary permeability, metabolic and mitochondrial dysfunction, oxidative stress, and activation of cytokines and inflammatory factors [1,2]. Transthoracic echocardiography is the main tool to early diagnose cardiac dysfunction in septic shock patients [3], but the use of complementary tools to improve the diagnose and treatment of these patients is much warranted.


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