Dysnatremias, disorders of plasma sodium concentration ([Na+]), are frequently encountered in daily clinical practice and pose a diagnostic and therapeutic challenge for many clinicians[1]. Rapid correction of dysnatremias is a major concern during treatment, as this might be accompanied by neurological complications such as central pontine myelinolysis in the case of hyponatremia and cerebral oedema in hypernatremic patients[2,3]. These complications are mainly a concern when plasma [Na+] alterations exceed eight mmol/L within 24 hours.
