High blood pressure (BP) is an important factor causing acute decompensated heart failure (HF). An abrupt increase in BP has been associated with a surge in neurohormonal and cytokine activation, which is one of the triggers of acute decompensated HF and reported to induce redistribution of fluids from the systemic to pulmonary circulation, further neurohormonal activation, and increased left ventricular (LV) afterload [1]. On the other hand, long-standing hypertension leads to structural remodeling of the heart [2,3].
