Obstructive sleep apnea (OSA) is a highly prevalent disorder characterized by recurrent upper airway collapse, intermittent hypoxemia, and sleep fragmentation [1]. Beyond its established cardiometabolic consequences, increasing evidence suggests a role for OSA in bone metabolism alterations [2,3]. However, the relationship between OSA severity and skeletal fragility remains inconsistent across studies, likely reflecting limitations of the apnea-hypopnea index (AHI) as a measure of disease burden [3,4].
